LoseWeightNYC
New member
On the subway this morning, I read an about a Mayo Clinic study suggesting that you can be at a “normal” weight for your height and still be considered “obese” by body fat percentage. The study went on to show that you are four times as likely to develop symptoms related to “metabolic syndrome” as someone who is at a normal weight and not obese by body fat percentage. If this surprises you, then you obviously don’t understand nutrition very well, particularly as it relates to health.
For a definition of terms, let’s define health as the absence of disease. The World Health Organization doesn’t like this definition because that means that someone born with a chronic condition is not “healthy,” but I don’t care what they think because they claim that obesity is caused by inactivity combined with diets high in saturated fat.
So how does this happen? How can you be at a normal weight but still be obese? Further, does this mean that if you just lost 50 lbs and are now at a normal weight for your height yet still have a very high percentage of body fat, you are still at a very high risk for diabetes, heart disease, etc? It’s funny because the biology of fat creation in the body is actually very well established in diabetes research, endocrinology, and various fields of animal research, and yet somehow this has not been communicated over to the obesity guys, mostly because the cause of obesity is “obvious,” it’s just about eating more calories than you burn.
To shed light on why this is not true, we need to also understand the definition of the word “cause.” According to wordnet, cause is “events that provide the generative force that is the origin of something.” Saying that eating more calories than you burn is the “cause” of obesity is like saying that eating more calories than he burns is the “cause” of a child growing taller. While eating more calories may be a necessary component of growing taller, it is actually growth hormone that is causing the growth, as well as the overeating. Likewise, obesity is driven by hormones as well, but in this case it is the hormone insulin, which is released in response to carbohydrates.
There are a number of groups in which we can observe obesity without very much eating. The Pima Indians had over 50% obesity while on government rations. In modern day Africa, obese mothers can be observed bringing their emaciated children to the hospital. On the flip side, there are a number of groups in which they eat a ridiculous amount of food, and yet do not get fat.
Back to how someone can be obese at a normal body weight. If you know anything about type 1 diabetes prior to insulin treatment, you know that people with this disease were completely unable to put on weight and would appear emaciated no matter how much they ate. They could be force fed calories and still starve to death. This is because insulin promotes fat storage. On the other end of the diabetes spectrum, people who have type 2 diabetes and get insulin treatments always put on weight while on insulin. There were a number of other diseases that were treated with insulin, and it was always associated with weight gain. John Nash, of “A Beautiful Mind” fame, was famously treated with insulin for his schizophrenia, and his weight gain on the treatment is well documented. Anorexia used to be treated with insulin, and while this is not a safe or smart thing to do, it did put weight on anorexics.
The problem with insulin is that it causes fatty acids in the blood stream to be stored as triglycerides in the adipose tissue. This storage causes a fatty acid deprivation in the rest of the cells of your body, which results in internal starvation and more eating, further elevating insulin levels and increasing the rate at which fatty acids are stored in the adipose tissue. I will restate that the science behind how this works is not up for debate, as it is accepted in multiple fields of human biology. What is up for debate (but not really) is that insulin levels get uniquely elevated from carbohydrates. If you want more information on any of this, I recommend checking out (REMOVED)
To finish off the point, you can cut calories all you want, and you might even lose weight, but if you’re not cutting carbs, your body will cannibalize your muscle tissue while holding onto that fat tissue that you’re trying to get rid of. Despite being at a normal weight, that fat tissue is still a significant risk factor. Furthermore, if you do cut carbs, your body cannot store up excess fat, no matter how much you eat.
For a definition of terms, let’s define health as the absence of disease. The World Health Organization doesn’t like this definition because that means that someone born with a chronic condition is not “healthy,” but I don’t care what they think because they claim that obesity is caused by inactivity combined with diets high in saturated fat.
So how does this happen? How can you be at a normal weight but still be obese? Further, does this mean that if you just lost 50 lbs and are now at a normal weight for your height yet still have a very high percentage of body fat, you are still at a very high risk for diabetes, heart disease, etc? It’s funny because the biology of fat creation in the body is actually very well established in diabetes research, endocrinology, and various fields of animal research, and yet somehow this has not been communicated over to the obesity guys, mostly because the cause of obesity is “obvious,” it’s just about eating more calories than you burn.
To shed light on why this is not true, we need to also understand the definition of the word “cause.” According to wordnet, cause is “events that provide the generative force that is the origin of something.” Saying that eating more calories than you burn is the “cause” of obesity is like saying that eating more calories than he burns is the “cause” of a child growing taller. While eating more calories may be a necessary component of growing taller, it is actually growth hormone that is causing the growth, as well as the overeating. Likewise, obesity is driven by hormones as well, but in this case it is the hormone insulin, which is released in response to carbohydrates.
There are a number of groups in which we can observe obesity without very much eating. The Pima Indians had over 50% obesity while on government rations. In modern day Africa, obese mothers can be observed bringing their emaciated children to the hospital. On the flip side, there are a number of groups in which they eat a ridiculous amount of food, and yet do not get fat.
Back to how someone can be obese at a normal body weight. If you know anything about type 1 diabetes prior to insulin treatment, you know that people with this disease were completely unable to put on weight and would appear emaciated no matter how much they ate. They could be force fed calories and still starve to death. This is because insulin promotes fat storage. On the other end of the diabetes spectrum, people who have type 2 diabetes and get insulin treatments always put on weight while on insulin. There were a number of other diseases that were treated with insulin, and it was always associated with weight gain. John Nash, of “A Beautiful Mind” fame, was famously treated with insulin for his schizophrenia, and his weight gain on the treatment is well documented. Anorexia used to be treated with insulin, and while this is not a safe or smart thing to do, it did put weight on anorexics.
The problem with insulin is that it causes fatty acids in the blood stream to be stored as triglycerides in the adipose tissue. This storage causes a fatty acid deprivation in the rest of the cells of your body, which results in internal starvation and more eating, further elevating insulin levels and increasing the rate at which fatty acids are stored in the adipose tissue. I will restate that the science behind how this works is not up for debate, as it is accepted in multiple fields of human biology. What is up for debate (but not really) is that insulin levels get uniquely elevated from carbohydrates. If you want more information on any of this, I recommend checking out (REMOVED)
To finish off the point, you can cut calories all you want, and you might even lose weight, but if you’re not cutting carbs, your body will cannibalize your muscle tissue while holding onto that fat tissue that you’re trying to get rid of. Despite being at a normal weight, that fat tissue is still a significant risk factor. Furthermore, if you do cut carbs, your body cannot store up excess fat, no matter how much you eat.
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