I was told you should inhale through the nose when doing bench presses. Does that apply to all lifts?
Inhaling while lifting
- Thread starter philsaigon
- Start date
something like to get the air into your upper chest, then exhale through the mouth.
If you are pushing yourself, breathing through the nose at any time will not really work.
The best is to take a large breath through your mouth. You get more air faster that way.
The body does not care if you breath in through your nose or mouth, one will not let more air into your "upper chest" any more than the other.
The best is to take a large breath through your mouth. You get more air faster that way.
The body does not care if you breath in through your nose or mouth, one will not let more air into your "upper chest" any more than the other.
I'm pretty sure that air coming in through the mouth or the nose ends up in the lungs either way...
I've heard that there are some gases in the back of your nose that makes the blood take up more oxygen when it gets breathed down to the lunges, but that's something I saw this doctor say on Oprah, so take it as you will 
I don't get the chest thing, when lifting you really want to breathe into your stomach to create a bigger internal pressure to help stabilizing you when you lift.
I don't get the chest thing, when lifting you really want to breathe into your stomach to create a bigger internal pressure to help stabilizing you when you lift.
As a general rule, your nose does a better job of filtering out debris than your mouth, so nose breathing is generally better than mouth breathing. But, under heavy exertion, you cannot get enough through your nose, at least I can't, so I use my mouth.
Karky, what do you mean by breathing into your stomach?
Karky, what do you mean by breathing into your stomach?
The main difference between breathing in your mouth vs your nose is that the nose will add more moisture to the air before it hits the lungs. While this can prevent your lungs burning during aerobic work in cold weather, it shouldn't matter while lifting. Also, the idea that inhaling in your mouth vs the nose has something to do with where air winds up is completely misguided.
Nasal breathing, nitric oxide, vasodilitation
Nasal breathing carries nitric oxide produced in the paranasal sinuses to the lungs, where it can have a vasodilating effect. See article abstract below.
Acta Physiologica Scandinavica
Volume 158 Issue 4 Page 343-347, December 1996
To cite this article: J. O. N LUNDBERG, G SETTERGREN, S GELINDER, J. M LUNDBERG, K ALVING, E WEITZBERG (1996)
Inhalation of nasally derived nitric oxide modulates pulmonary function in humans
Acta Physiologica Scandinavica 158 (4), 343–347.
doi:10.1046/j.1365-201X.1996.557321000.x
Prev Article Next Article
Abstract
Inhalation of nasally derived nitric oxide modulates pulmonary function in humans
J. O. N. LUNDBERG, G. SETTERGREN, S. GELINDER, J. M. LUNDBERG, K. ALVING & E. WEITZBERG1Department of Physiology and Pharmacology, Karolinska Institute, Stockholm, Sweden2Department of Cardiothoracic Anaesthetics and Intensive Care, Karolinska Institute, Stockholm, Sweden3Anaesthesiology and Intensive Care, Karolinska Hospital, Stockholm, Sweden
Correspondence to: J. O. N. Lundberg Department of Physiology and Pharmacology, Karolinska Institute, 171 77 Stockholm, Sweden.
This study was supported by grants from the Swedish Medical Research Council, the Swedish Tobacco Company, the Magnus Bergwall Foundation, the Åke Wiberg Foundation, AGA AB Medical Research Fund, the Swedish Environmental Health Agency, the Tore Nilson Foundation for Medical Research.
Abstract
The vasodilator gas nitric oxide (NO) is produced in the paranasal sinuses and is excreted continuously into the nasal airways of humans. This NO will normally reach the lungs with inspiration, especially during nasal breathing. We wanted to investigate the possible effects of low-dose inhalation of NO from the nasal airways on pulmonary function. The effects of nasal and oral breathing on transcutaneous oxygen tension (tcPO2) were studied in healthy subjects. Furthermore, we also investigated whether restoring low-dose NO inhalation would influence pulmonary vascular resistance index (PVRI) and arterial oxygenation (PaO2) in intubated patients who are deprived of NO produced in the nasal airways. Thus, air derived from the patient's own nose was aspirated and led into the inhalation limb of the ventilator. In six out of eight healthy subjects tcPO2 was 10% higher during periods of nasal breathing when compared with periods of oral breathing. In six out of six long-term intubated patients PaO2 increased by 18% in response to the addition of nasal air samples. PVRI was reduced by 11% in four of 12 short-term intubated patients when nasal air was added to the inhaled air. The present study demonstrates that tcPO2 increases during nasal breathing compared with oral breathing in healthy subjects. Furthermore, in intubated patients, who are deprived of self-inhalation of endogenous NO, PaO2 increases and pulmonary vascular resistance may decrease by adding NO-containing air, derived from the patient's own nose, to the inspired air. The involvement of self-inhaled NO in the regulation of pulmonary function may represent a novel physiological principle, namely that of an enzymatically produced airborne messenger. Furthermore, our findings may help to explain one biological role of the human paranasal sinuses.
Nasal breathing carries nitric oxide produced in the paranasal sinuses to the lungs, where it can have a vasodilating effect. See article abstract below.
Acta Physiologica Scandinavica
Volume 158 Issue 4 Page 343-347, December 1996
To cite this article: J. O. N LUNDBERG, G SETTERGREN, S GELINDER, J. M LUNDBERG, K ALVING, E WEITZBERG (1996)
Inhalation of nasally derived nitric oxide modulates pulmonary function in humans
Acta Physiologica Scandinavica 158 (4), 343–347.
doi:10.1046/j.1365-201X.1996.557321000.x
Prev Article Next Article
Abstract
Inhalation of nasally derived nitric oxide modulates pulmonary function in humans
J. O. N. LUNDBERG, G. SETTERGREN, S. GELINDER, J. M. LUNDBERG, K. ALVING & E. WEITZBERG1Department of Physiology and Pharmacology, Karolinska Institute, Stockholm, Sweden2Department of Cardiothoracic Anaesthetics and Intensive Care, Karolinska Institute, Stockholm, Sweden3Anaesthesiology and Intensive Care, Karolinska Hospital, Stockholm, Sweden
Correspondence to: J. O. N. Lundberg Department of Physiology and Pharmacology, Karolinska Institute, 171 77 Stockholm, Sweden.
This study was supported by grants from the Swedish Medical Research Council, the Swedish Tobacco Company, the Magnus Bergwall Foundation, the Åke Wiberg Foundation, AGA AB Medical Research Fund, the Swedish Environmental Health Agency, the Tore Nilson Foundation for Medical Research.
Abstract
The vasodilator gas nitric oxide (NO) is produced in the paranasal sinuses and is excreted continuously into the nasal airways of humans. This NO will normally reach the lungs with inspiration, especially during nasal breathing. We wanted to investigate the possible effects of low-dose inhalation of NO from the nasal airways on pulmonary function. The effects of nasal and oral breathing on transcutaneous oxygen tension (tcPO2) were studied in healthy subjects. Furthermore, we also investigated whether restoring low-dose NO inhalation would influence pulmonary vascular resistance index (PVRI) and arterial oxygenation (PaO2) in intubated patients who are deprived of NO produced in the nasal airways. Thus, air derived from the patient's own nose was aspirated and led into the inhalation limb of the ventilator. In six out of eight healthy subjects tcPO2 was 10% higher during periods of nasal breathing when compared with periods of oral breathing. In six out of six long-term intubated patients PaO2 increased by 18% in response to the addition of nasal air samples. PVRI was reduced by 11% in four of 12 short-term intubated patients when nasal air was added to the inhaled air. The present study demonstrates that tcPO2 increases during nasal breathing compared with oral breathing in healthy subjects. Furthermore, in intubated patients, who are deprived of self-inhalation of endogenous NO, PaO2 increases and pulmonary vascular resistance may decrease by adding NO-containing air, derived from the patient's own nose, to the inspired air. The involvement of self-inhaled NO in the regulation of pulmonary function may represent a novel physiological principle, namely that of an enzymatically produced airborne messenger. Furthermore, our findings may help to explain one biological role of the human paranasal sinuses.
This is a good question! Everyone may have their own thought. This is what I have experienced! Always breath through the nose while taking the air in! In all the lifts! Holding the breath through the range of motion till above the midway point, then slowly start exhaling through the moth to end of the lift! Reason being is not to get any air into the stomach. Breathing through the moth you have a greater risk of getting air into the stomach. You want to fill up the chest cavity! Look at it this way. Lets say your body is like a balloon! You take the air in through the nose to fill the lungs, as not to get any in your stomach! More air in the lungs gives you a more solid chest and upper cavity! Just like filling a balloon up with air. Now that the balloon is full, Holding it in throughout the range of motion will help keep you solid! If you was to start letting the air out before you are above the midway point you may run into a problem! Think of this! If you was to fill up a balloon, pinch the end of the balloon as like holding your breath, then have pressure put on the balloon, you will see that the balloon is still solid, with pressure. Letting any air out before the midway point would start to deflate the balloon. The balloon would be no longer solid and the pressure of the weight would collapse the balloon. You would have no pushing power left! This is why I do it this way! As to control the weight through the full range of motion without running out of gas and power if you will! Again, this is just what I have experienced!
God Bless!
Franklin McQuaid
God Bless!
Franklin McQuaid
that's what I've heard too, MDfitnessfan! thanks for that.
by breating into your stomach I really mean breathe with your stomach. Blow your stomach out to suck air in. It helps with intra abdominal preassure.
however, you can get more air in if you breathe with your shoulders (the "expand the chest" breathing) which is why you'll probably do that more if you're running or something like that.
by breating into your stomach I really mean breathe with your stomach. Blow your stomach out to suck air in. It helps with intra abdominal preassure.
however, you can get more air in if you breathe with your shoulders (the "expand the chest" breathing) which is why you'll probably do that more if you're running or something like that.
This is exactly what the guy that told me said (except w/out all the exclamation marks
)I breathe in mostly through my nose as this is simply a common way to control and measure your breathing, but I wouldn't think breathing through your mouth is a problem.
...A lot of the time during squats or deads I still inhale through my nose, but I have to take a few gasps through my mouth in between reps
...A lot of the time during squats or deads I still inhale through my nose, but I have to take a few gasps through my mouth in between reps